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She's still at it

San Diego woman wants half of $100K raised for Starbucks barista following mask dispute

"Now, Gilles believes she is entitled to half of the cash, KGTV reported Tuesday.

"It was discrimination, and everybody is OK with it and enabling and rewarding that behavior," she told the news outlet.

Gilles, who previously told KNSD that she is considering suing Cowan, added that she has contacted attorneys, but they all charge too much. She said she hopes to raise money with her own crowdfunding campaign, KGTV reported.

Gilles also claimed that she can't wear a mask because she experiences symptoms such as shortness of breath, dizziness, asthma and mask acne, according to KGTV. She went on to show KGTV documentation from a 2015 pelvic exam, as well as a note from a chiropractor saying that Gilles "has underlying breath conditions that prevent her from wearing a mask." The chiropractor declined to discuss Gilles with the news station."

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How deluded do you have to be to think any of that money should or will be yours?

Well I suspect from a technical psychiatric standpoint all this is not about delusions it's about severe character disorder. That's what set the stage for this whole confrontation in the first place, and personality being one of the great and wonderfully predictable constants in the social universe always reveals itself. Over and over.
 
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Screen Shot 2020-07-15 at 6.51.59 PM.png



https://twitter.com/AVG_Joseph96/status/1283234509639618560?s=20
 
'the wire' is an amazing show and has gotton to be a must-watch for pop culture references.

it seriously is must-watch. it says a lot, in that show. and its entertaining - coming from a person who can't stand most tv and even most modern movies.

The Wire was hard to watch sometimes, it's excellently done, but it shows a window into a grim world. The decent of that school kid was one of those to difficult to watch.
 
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Ughh... Sad to report that in a county that has reported at most one death a day (and few and far between until recently), today we had four...... And the info I posted a few days ago about the man/woman passing in their late 30s/early 40s never came out.... It was said that they were not from our county and just left at that. Found out today that one office manager had it since last Wednesday and was still coming into work... He left yesterday having trouble taking a full breath. He's in his late 30s but very overweight. Luckily I work out on the road so no direct exposure.


The Wire was hard to watch sometimes, it's excellently done, but it shows a window into a grim world. The decent of that school kid was one of those to difficult to watch.
'You gonna look out for me? You mean it? You're gonna look out for me? You promise?'

disturbs the soul.......
 
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It’s been so short time since COVID-19 actually started, that I’m curious whether an actual scientific study has yet been conducted (with volunteers, for example, not with just test tubes), about the actual contagiousness and seriouness of COVID-19, or is it all still just a bunch of guesswork and anecdotal (might not actually be causal, just correlated) evidence and rumors from all over the place? I mean rigorious testing in various circumstances:
  • person-to-person, open air at various distances (with and without different levels of wind), with and without mask, different kinds of masks, with coffing, without coffing or sneezing
  • person-to-person, cubicle, with and without mask, different kinds of masks, with coffing, without coffing or sneezing
  • person-to-person, inhouse in typical shop or restaurant setting, with and without mask, different kinds of masks, with coffing, without coffing or sneezing
  • person-to-person, where-have-you, with and without mask, different kinds of masks, with coffing, without coffing or sneezing
  • contracting from touching contaminated PPE
  • contracting by hands from various sources, public places (buttons, handles, plastic, other materials, with or/without sunshine, after various intervals)
And furthermore, the effect of covid on people with different backgrounds:
  • healthy, different age groups, different treatment protocols, including analysis of any residual long-lasting effects per group
  • different pre-existing conditions, different age groups, different treatment protocols, including analysis of any residual long-lasting effects per group
It seems that simply backtracing and trying to deduce the above from non-pre-coordinated random cases leads to a whole lot of innuendo. If all the above was done in a pre-coordinated (and reproducible; you could even re-(don't)-try-it-at-home, if you wanted) way, even Foxnews audience could not deny it.
 
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It’s been so short time since COVID-19 actually started, that I’m curious whether an actual scientific study has yet been conducted (with volunteers, for example, not with just test tubes), about the actual contagiousness and seriouness of COVID-19, or is it all still just a bunch of guesswork and anecdotal (might not actually be causal, just correlated) evidence and rumors from all over the place? I mean rigorious testing in various circumstances:
  • person-to-person, open air at various distances (with and without different levels of wind), with and without mask, different kinds of masks, with coffing, without coffing or sneezing
  • person-to-person, cubicle, with and without mask, different kinds of masks, with coffing, without coffing or sneezing
  • person-to-person, inhouse in typical shop or restaurant setting, with and without mask, different kinds of masks, with coffing, without coffing or sneezing
  • person-to-person, where-have-you, with and without mask, different kinds of masks, with coffing, without coffing or sneezing
  • contracting from touching contaminated PPE
  • contracting by hands from various sources, public places (buttons, handles, plastic, other materials, with or/without sunshine, after various intervals)
And furthermore, the effect of covid on people with different backgrounds:
  • healthy, different age groups, different treatment protocols, including analysis of any residual long-lasting effects per group
  • different pre-existing conditions, different age groups, different treatment protocols, including analysis of any residual long-lasting effects per group
It seems that simply backtracing and trying to deduce the above from non-pre-coordinated random cases leads to a whole lot of innuendo. If all the above was done in a pre-coordinated (and reproducible; you could even re-(don't)-try-it-at-home, if you wanted) way, even Foxnews audience could not deny it.
Are you volunteering?
 

Just a bit.

41577_2016_Article_BFnri201510_Fig1_HTML.jpg


Following infection, T cells differentiate into a heterogeneous population of effector T cells that can mediate pathogen clearance. A subset of these effector T cells possesses the ability to survive long term and mature into memory T cells that can provide long-term immunity. Understanding the signals that regulate the development of memory T cells is crucial to efforts to design vaccines capable of eliciting T cell-based immunity. CD4+ T cells are essential in the formation of protective memory CD8+ T cells following infection or immunization. However, until recently, the mechanisms by which CD4+ T cells act to support memory CD8+ T cell development following infection were unclear. Here, we discuss recent studies that provide insight into the multifaceted role of CD4+ T cells in the regulation of memory CD8+ T cell differentiation.

Key Points
  • Following immunization, CD4+ T cells promote the induction of a robust primary CD8+ T cell response through numerous mechanisms, including licensing of dendritic cells (DCs) and promoting the interaction between DCs and CD8+ T cells.

  • CD4+ T cells regulate the secondary responsiveness of CD8+ T cells during immunization through suppression of TNF-related apoptosis-inducing ligand (TRAIL) through a process dependent on licensing of DCs to produce interleukin-15 (IL-15) and autocrine secretion of IL-2 by CD8+ T cells.

  • Following infection, CD4+ T cell help is necessary for the induction of a memory CD8+ T cell pool capable of mediating protective immunity but is largely dispensable for a robust primary response.

  • Regulatory T (TReg) cells act during the resolution phase of infection to protect CD8+ T cells from inflammatory signals and promote the survival of a CD8+ T cell pool capable of robustly expanding upon secondary infection.

  • CD4+ T cell help promotes the induction of tissue-resident memory CD8+ T cells during mucosal infection through guidance of CD8+ T cells into a microenvironment where they can become exposed to the signals necessary for their continued maintenance within the tissue.

  • During chronic infection, effector CD4+ T cells support the maintenance of functional CD8+ T cells through secretion of IL-21, whereas TReg cells dampen the CD8+ T cell response through suppression of DCs.

  • Quiz at 11 - if you pass, then you can argue (or even just have a discussion) with Dr Fauci! If you think this is impenetrable, welcome to the humbling complexity of (our) Nature.
It should be obvious that infectious disease management should be left to people that understand immunity and infection.
 
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It’s been so short time since COVID-19 actually started, that I’m curious whether an actual scientific study has yet been conducted (with volunteers, for example, not with just test tubes), about the actual contagiousness and seriouness of COVID-19, or is it all still just a bunch of guesswork and anecdotal (might not actually be causal, just correlated) evidence and rumors from all over the place? I mean rigorious testing in various circumstances:
  • person-to-person, open air at various distances (with and without different levels of wind), with and without mask, different kinds of masks, with coffing, without coffing or sneezing
  • person-to-person, cubicle, with and without mask, different kinds of masks, with coffing, without coffing or sneezing
  • person-to-person, inhouse in typical shop or restaurant setting, with and without mask, different kinds of masks, with coffing, without coffing or sneezing
  • person-to-person, where-have-you, with and without mask, different kinds of masks, with coffing, without coffing or sneezing
  • contracting from touching contaminated PPE
  • contracting by hands from various sources, public places (buttons, handles, plastic, other materials, with or/without sunshine, after various intervals)
And furthermore, the effect of covid on people with different backgrounds:
  • healthy, different age groups, different treatment protocols, including analysis of any residual long-lasting effects per group
  • different pre-existing conditions, different age groups, different treatment protocols, including analysis of any residual long-lasting effects per group
It seems that simply backtracing and trying to deduce the above from non-pre-coordinated random cases leads to a whole lot of innuendo. If all the above was done in a pre-coordinated (and reproducible; you could even re-(don't)-try-it-at-home, if you wanted) way, even Foxnews audience could not deny it.

You clearly don't do science. Every test probe on your first set of parameters would be seriously unethical to test in the real world with an active COVID-19 patient, let alone at scale, which would be required to derive statistics that weren't 'anecdotal' or single case, as you say. But we have plenty of information proving that COVID is highly contagious, such that we don't need more, unless of course, you believe that the virus is just being hyped and it's all a big hoax - just a big lie to prevent our Great President from being re elected. If you think there's a chance of that, we have bigger problems here than explaining medical ethics.

Given the enormous evidence that COVID-19 spreads quickly and easily when people are exposed to someone actively infected but not socially distanced or wearing masks, your set of probes would be roughly equivalent to someone wanting more proof that the 4000 lb granite boulder that landed on their car after falling off a cliff actually did all the damage, so, in the interests of science and the avoidance of anecdotal reasoning, they bought a new car, and dropped another 4000 lb boulder on it from a helicopter to confirm that the damage attributed to the first impact was in fact real.

In this case, Florida, Texas, Arizona, and about 35 other states have already done the 'drop-the-boulder-on-the-car-a-second-time' experiment. We are witnessing the results. How do think it's going in those states, that all opened up and operated from the Trump playbook, that this thing is A) no big deal; B) not that contagious. How is it going in all those states in your opinion??

How much more proof do you really need? And if you need more proof, is the problem a lack of clear evidence, as you seem to imply, or is it simply impaired reality testing. I'd say it's probably the latter.

As for the second set of questions, there is a lot of data and a growing body of information on that already. Just look at it with your eyes open.
 
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