SMAlset
Well-Known Member
I don't get to come here every day to read all the new pages but have you guys seen any additional studies on asymptomatic individuals' lung scans? I've seen a few articles indicating they have shown up with white, glassy spots but that's about it. Assume that the majority of the people never make it to a hospital or doctor setting for a scan.
Found this article from NPR (6/23/20) that indicated covid damage had occurred in their lungs to some extent and "likely" will be reversable. Curious if that "reversable" assumption has panned out to be true or not through longer term studies. If not, would it make you more susceptible to the next virus round or regular old pneumonia etc., kind of putting them in the more at risk category going forward?
We Still Don't Fully Understand The Label 'Asymptomatic'
Today the Washington Post had this article on autopsies done on brains, lungs and hearts and the pathologist in the article found some interesting, unexpected results and a similarity to dengue.
https://www.washingtonpost.com/health/2020/07/01/coronavirus-autopsies-findings/
From Washington Post article on examining the brain and on oxygen levels:
Found this article from NPR (6/23/20) that indicated covid damage had occurred in their lungs to some extent and "likely" will be reversable. Curious if that "reversable" assumption has panned out to be true or not through longer term studies. If not, would it make you more susceptible to the next virus round or regular old pneumonia etc., kind of putting them in the more at risk category going forward?
We Still Don't Fully Understand The Label 'Asymptomatic'
Today the Washington Post had this article on autopsies done on brains, lungs and hearts and the pathologist in the article found some interesting, unexpected results and a similarity to dengue.
https://www.washingtonpost.com/health/2020/07/01/coronavirus-autopsies-findings/
From Washington Post article on examining the brain and on oxygen levels:
"He found snippets of virus in only some areas, and it was unclear whether they were dead remnants or active virus when the patient died. There were only small pockets of inflammation. But there were large swaths of damage due to oxygen deprivation. Whether the deceased were longtime intensive care patients or people who died suddenly, Solomon said, the pattern was eerily similar.
“We were very surprised,” he said.
When the brain does not get enough oxygen, individual neurons die, and that death is permanent. To a certain extent, people’s brains can compensate, but at some point, the damage is so extensive that different functions start to degrade.
On a practical level, Solomon said, if the virus is not getting into the brain in large amounts, that helps with drug development because treatment becomes trickier when it is pervasive, for instance, in some patients with West Nile or HIV. Another takeaway is that the findings underscore the importance of getting people on supplementary oxygen quickly to prevent irreversible damage."
“We were very surprised,” he said.
When the brain does not get enough oxygen, individual neurons die, and that death is permanent. To a certain extent, people’s brains can compensate, but at some point, the damage is so extensive that different functions start to degrade.
On a practical level, Solomon said, if the virus is not getting into the brain in large amounts, that helps with drug development because treatment becomes trickier when it is pervasive, for instance, in some patients with West Nile or HIV. Another takeaway is that the findings underscore the importance of getting people on supplementary oxygen quickly to prevent irreversible damage."
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